Antrax

Antrax is a rough infectious disease caused by Bacillus anthracis, a positive gram bacteria, aerobe formed from endospores, encapsulated, that can be half ordinary cultivated. The vegetative cell is large (1-8 microns in longitude, 1-1.5 microns wide). The size of a spore is approximately 1 micron. Spores grow rapidly in all cultivated ordinary labs at 37degrees celcius. Its cell morphology and colonial allows its identification by an experienced microbiologist, even though fe

w microbiologists outside of the vetinary community have seen antrax colonies more than in text books. Human and animal death frequently an experimentaly occurs suddenly and unexpectedly. From these observations it is concluded that exotoxin plays an important role in the pathogeny of antrax. anthracis were not recognized until 1954. Etymology: Anthracis originated from the Greek word for carbon, anthrakis, because the disease causes black superficial injuries like carbon. Cepas: There are two even varient colonial(s) a rough(r) that are related with the ability to form a capsule. An antrax toxin component has a deadly action form that at this time is not discovered. Besides, the animals plasmic infection cells contain a toxic that causes antrax symptoms when it is injected in normal rabbits. -PathogencyThe toxic properties of B. The three virulence factores are: the edema toxin, the deadly toxin and a capsular antigenic. The R varients are relativaly avirulent. The capsule is not toxic, it acts as protectionj against phagocytosis and it plays its most important role during the establishment of the disease, and a less significant role in the last phase of the disease that is measured by antrax toxin. Previously, due to the tremendous number of observed bacteria in infected animal blood (109 bacteria ml), it was supposed that death was due to the capillary obstruction,But experimentaly it was demonstrated that only approximately 3 million cellsml are necessary to cause the animals death. Death is apparantly due to the lack of oxygen, secondary shock, gained vascular permeability, respitory failure and cardio failure.