in the androgen receptor gene have been associated with a higher incidence of prostate cancer. While a moderate expansion of the CAG repeat region has a modulating effect on androgen-receptor function, whereas any expansion beyond a threshold is likely to trigger separate processes. This difference makes it difficult to identify a specific phenotype that is associated with the expansion of CAG repeat length. A very uncommon identifiable phenotype is testicular size and hormone concentration (Dowsing AT, et al., 1999).
             Analyses of trinucleotide (CAG) repeat length and point mutations in the androgen-receptor gene are done by PCR, single-stranded conformational polymorphism, and DNA sequencing. When 32 controls and 35 infertile men were screened for androgen- receptor gene mutations, the genomic DNA samples were coded, an
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